Specialized Function of Tissues of the Body

Amino acids can also be degraded to acetyl-CoA by the liver for use by the nous during famishment.

Low blood sugar levels which result from famishment can cause irreversible brain damage (Andersen; Integration). When starvation occurs, the brain has to use ketone bodies as a source of acetyl-CoA. It uses its efficacy to maintain an electrical potential in the neurons so they bear upon to function. In starvation, neuronal signals from the brain induce the release of adrenaline from the adrenal medulla, the normal response to stress. This results in glycogen phosphorylate energizing and inactivation of glycogen synthase, i.e. glucose storage is inhibited and its release into the bloodstream promoted by the segmentation of glycogen stored in the liver. Glucagon secretion is induce and insulin secretion inhibited since insulin lowers the blood sugar level. Anaerobic breakdown of glycogen in skeletal muscle is also stimulated. change mobilization from adipose tissue is stimulated t

Medical Information. "Possible complications of eating disorders." 2005. 3 Sep. 2005.

over the next few days, oxaloacetate is depleted in reservation glucose and leave an accumulation of acetyl-CoA, which favors the formation of ketone bodies by the liver (Integration).

The brain uses these as fuel in place of glucose. When fat militia atomic number 18 depleted, the body begins breaking down essential proteins, which leads to release of liver and heart function as these organs are broken down for fuel. Triacylglycerols would theoretically let enough power for the body for months, but would lead to morbidly high levels of ketone bodies.

hrough cAMP-mediated phosphorylation to provide ketone bodies for acetyl-CoA for the starving brain.

Brain starvation can be right by returning food using up to normal (Medical). However, during a period of starvation, the gastrointestinal tract shrinks in size and cannot defend the same amount of food as before. Food intake must be increased slowly,
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